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  1. Neuromyelitis optica/spectrum disorder (NMO/SD) is a severe, inflammatory disease of the central nervous system (CNS). In the majority of patients, it is associated with the presence of pathogenic serum autoan...

    Authors: Bleranda Zeka, Maria Hastermann, Nathalie Kaufmann, Kathrin Schanda, Marko Pende, Tatsuro Misu, Paulus Rommer, Kazuo Fujihara, Ichiro Nakashima, Charlotte Dahle, Fritz Leutmezer, Markus Reindl, Hans Lassmann and Monika Bradl
    Citation: Acta Neuropathologica Communications 2016 4:82
  2. Functional and molecular changes associated with pathophysiological conditions are relatively easily detected based on tissue samples collected from patients. Population specific cellular responses to disease ...

    Authors: Nóra Faragó, Ágnes Katalin Kocsis, Csilla Braskó, Sándor Lovas, Márton Rózsa, Judith Baka, Balázs Kovács, Katalin Mikite, Viktor Szemenyei, Gábor Molnár, Attila Ozsvár, Gáspár Oláh, Ildikó Piszár, Ágnes Zvara, Attila Patócs, Pál Barzó…
    Citation: Acta Neuropathologica Communications 2016 4:78
  3. Amyotrophic lateral sclerosis (ALS) is characterised by the death of upper (corticospinal) and lower motor neurons (MNs) with progressive muscle weakness. This incurable disease is clinically heterogeneous and...

    Authors: Matthew J. Fogarty, Erica W. H. Mu, Peter G. Noakes, Nickolas A. Lavidis and Mark C. Bellingham
    Citation: Acta Neuropathologica Communications 2016 4:77
  4. Vascular cognitive impairment is the second most common form of dementia. The pathogenic pathways leading to vascular cognitive impairment remain unclear but clinical and experimental data have shown that chro...

    Authors: Raman Saggu, Toni Schumacher, Florian Gerich, Cordula Rakers, Khalid Tai, Andrea Delekate and Gabor C. Petzold
    Citation: Acta Neuropathologica Communications 2016 4:76
  5. Recent research findings have provided convincing evidence indicating a role for Interleukin-33 (IL-33) signalling pathway in a number of central nervous system (CNS) diseases including multiple sclerosis (MS)...

    Authors: Debbie Allan, Karen J. Fairlie-Clarke, Christina Elliott, Cornelia Schuh, Susan C. Barnett, Hans Lassmann, Christopher Linnington and Hui-Rong Jiang
    Citation: Acta Neuropathologica Communications 2016 4:75
  6. Reduction of Tau protein expression was described in 2003 by Zhukareva et al. in a variant of frontotemporal lobar degeneration (FTLD) referred to as diagnosis of dementia lacking distinctive histopathology, t...

    Authors: Anthony Papegaey, Sabiha Eddarkaoui, Vincent Deramecourt, Francisco-Jose Fernandez-Gomez, Pierre Pantano, Hélène Obriot, Camille Machala, Vincent Anquetil, Agnès Camuzat, Alexis Brice, Claude-Alain Maurage, Isabelle Le Ber, Charles Duyckaerts, Luc Buée, Nicolas Sergeant and Valérie Buée-Scherrer
    Citation: Acta Neuropathologica Communications 2016 4:74
  7. Human BK polyomavirus (BKV) is reactivated under conditions of immunosuppression leading most commonly to nephropathy or cystitis; its tropism for the brain is rare and poorly understood. We present a unique c...

    Authors: Armine Darbinyan, Eugene O. Major, Susan Morgello, Steven Holland, Caroline Ryschkewitsch, Maria Chiara Monaco, Thomas P. Naidich, Joshua Bederson, Joanna Malaczynska, Fei Ye, Ronald Gordon, Charlotte Cunningham-Rundles, Mary Fowkes and Nadejda M. Tsankova
    Citation: Acta Neuropathologica Communications 2016 4:73
  8. A neuro-inflammatory response is evident in Alzheimer’s disease (AD), yet the precise mechanisms by which neuro-inflammation influences the progression of Alzheimer’s disease (AD) remain poorly understood. Typ...

    Authors: Myles R. Minter, Zachery Moore, Moses Zhang, Kate M. Brody, Nigel C. Jones, Sandy R. Shultz, Juliet M. Taylor and Peter J. Crack
    Citation: Acta Neuropathologica Communications 2016 4:72
  9. Next generation sequencing (NGS) can globally interrogate the genetic composition of biological samples in an unbiased yet sensitive manner. The objective of this study was to utilize the capabilities of NGS t...

    Authors: Michael J. Strong, Eugene Blanchard 4th, Zhen Lin, Cindy A. Morris, Melody Baddoo, Christopher M. Taylor, Marcus L. Ware and Erik K. Flemington
    Citation: Acta Neuropathologica Communications 2016 4:71
  10. Amyotrophic Lateral Sclerosis (ALS) is the most frequent motor neuron disease in adults. Classical ALS is characterized by the death of upper and lower motor neurons leading to progressive paralysis. Approxima...

    Authors: Vincent Picher-Martel, Paul N. Valdmanis, Peter V. Gould, Jean-Pierre Julien and Nicolas Dupré
    Citation: Acta Neuropathologica Communications 2016 4:70
  11. Alexander disease is a fatal neurological illness characterized by white-matter degeneration and formation of Rosenthal fibers, which contain glial fibrillary acidic protein as astrocytic inclusion. Alexander ...

    Authors: Takayuki Kondo, Misato Funayama, Michiyo Miyake, Kayoko Tsukita, Takumi Era, Hitoshi Osaka, Takashi Ayaki, Ryosuke Takahashi and Haruhisa Inoue
    Citation: Acta Neuropathologica Communications 2016 4:69

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:101

  12. Multiple sclerosis is a devastating neurological disorder characterized by the autoimmune destruction of the central nervous system myelin. While T cells are known orchestrators of the immune response leading ...

    Authors: Tzu-Ying Chuang, Yong Guo, Scott M. Seki, Abagail M. Rosen, David M. Johanson, James W. Mandell, Claudia F. Lucchinetti and Alban Gaultier
    Citation: Acta Neuropathologica Communications 2016 4:68
  13. Notch1 signaling is a cellular cascade with a fundamental role from brain development to adult brain function. Reduction in Notch1 affects synaptic plasticity, memory and olfaction. On the other hand, Notch1 o...

    Authors: Emanuele Brai, Noemi Alina Raio and Lavinia Alberi
    Citation: Acta Neuropathologica Communications 2016 4:64

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:90

  14. Dementia with Lewy bodies (DLB) patients frequently experience well formed recurrent complex visual hallucinations (RCVH). This is associated with reduced blood flow or hypometabolism on imaging of the primary...

    Authors: Ahmad A. Khundakar, Peter S. Hanson, Daniel Erskine, Nichola Z. Lax, Joseph Roscamp, Evangelia Karyka, Eliona Tsefou, Preeti Singh, Simon J. Cockell, Andrew Gribben, Lynne Ramsay, Peter G. Blain, Urs P. Mosimann, Deborah J. Lett, Matthias Elstner, Douglass M. Turnbull…
    Citation: Acta Neuropathologica Communications 2016 4:66
  15. Inflammation is becoming increasingly recognized as an important contributor to Alzheimer’s disease (AD) pathogenesis. As a part of the innate immune system, the complement cascade enhances the body’s ability ...

    Authors: Luke W. Bonham, Rahul S. Desikan and Jennifer S. Yokoyama
    Citation: Acta Neuropathologica Communications 2016 4:65
  16. Alzheimer disease is characterized by the accumulation of β-amyloid (Aβ) plaques and tau-laden neurofibrillary tangles. Emerging studies suggest that in neurodegenerative diseases, aggregation of one protein s...

    Authors: Wesley Chen, Edsel A. Abud, Stephen T. Yeung, Anita Lakatos, Trevor Nassi, Jane Wang, David Blum, Luc Buée, Wayne W. Poon and Mathew Blurton-Jones
    Citation: Acta Neuropathologica Communications 2016 4:63
  17. Common neurodegenerative proteinopathies, such as Alzheimer’s disease (AD) and Parkinson’s disease (PD), are characterized by the misfolding and aggregation of toxic protein species, including the amyloid beta...

    Authors: Amit K. Chouhan, Caiwei Guo, Yi-Chen Hsieh, Hui Ye, Mumine Senturk, Zhongyuan Zuo, Yarong Li, Shreyasi Chatterjee, Juan Botas, George R. Jackson, Hugo J. Bellen and Joshua M. Shulman
    Citation: Acta Neuropathologica Communications 2016 4:62
  18. Frontotemporal lobar degeneration (FTLD) and amyotrophic lateral sclerosis (ALS) are types of major TDP-43 (43-kDa TAR DNA-binding protein) proteinopathy. Cortical TDP-43 pathology has been analyzed in detail ...

    Authors: Ryoko Takeuchi, Mari Tada, Atsushi Shiga, Yasuko Toyoshima, Takuya Konno, Tomoe Sato, Hiroaki Nozaki, Taisuke Kato, Masao Horie, Hiroshi Shimizu, Hirohide Takebayashi, Osamu Onodera, Masatoyo Nishizawa, Akiyoshi Kakita and Hitoshi Takahashi
    Citation: Acta Neuropathologica Communications 2016 4:61
  19. Gliomas are the most frequent intraaxial CNS neoplasms with a heterogeneous molecular background. Recent studies on diffuse gliomas have shown frequent alterations in the genes involved in chromatin remodellin...

    Authors: Azadeh Ebrahimi, Marco Skardelly, Irina Bonzheim, Ines Ott, Helmut Mühleisen, Franziska Eckert, Ghazaleh Tabatabai and Jens Schittenhelm
    Citation: Acta Neuropathologica Communications 2016 4:60
  20. It is essential to determine the specificity of AV-1451 PET for tau in brain imaging by using pathological comparisons. We performed autoradiography in autopsy-confirmed Alzheimer disease and other neurodegene...

    Authors: Val J. Lowe, Geoffry Curran, Ping Fang, Amanda M. Liesinger, Keith A. Josephs, Joseph E. Parisi, Kejal Kantarci, Bradley F. Boeve, Mukesh K. Pandey, Tyler Bruinsma, David S. Knopman, David T. Jones, Leonard Petrucelli, Casey N. Cook, Neill R. Graff-Radford, Dennis W. Dickson…
    Citation: Acta Neuropathologica Communications 2016 4:58
  21. Tissue invasion and infiltration by brain tumours poses a clinical challenge, with destruction of structures leading to morbidity. We assessed whether micro-CT could be used to map tumour invasion in adamantin...

    Authors: John R. Apps, J. Ciaran Hutchinson, Owen J. Arthurs, Alex Virasami, Abhijit Joshi, Berit Zeller-Plumhoff, Dale Moulding, Thomas S. Jacques, Neil J. Sebire and Juan Pedro Martinez-Barbera
    Citation: Acta Neuropathologica Communications 2016 4:57
  22. Non-syndromic pituitary gigantism can result from AIP mutations or the recently identified Xq26.3 microduplication causing X-linked acrogigantism (XLAG). Within Xq26.3, GPR101 is believed to be the causative gene...

    Authors: Donato Iacovazzo, Richard Caswell, Benjamin Bunce, Sian Jose, Bo Yuan, Laura C. Hernández-Ramírez, Sonal Kapur, Francisca Caimari, Jane Evanson, Francesco Ferraù, Mary N. Dang, Plamena Gabrovska, Sarah J. Larkin, Olaf Ansorge, Celia Rodd, Mary L. Vance…
    Citation: Acta Neuropathologica Communications 2016 4:56
  23. The adaptor protein NHERF1 (Na/H exchanger-3 regulatory factor-1) and its associated ezrin-radixin-moesin-merlin/neurofibromin-2 (ERM-NF2) family proteins are required for epithelial morphogenesis and have bee...

    Authors: Maria-Magdalena Georgescu, Bret C. Mobley, Brent A. Orr, Ping Shang, Norman L. Lehman, Xiaoping Zhu, Thomas J. O’Neill, Veena Rajaram, Kimmo J. Hatanpaa, Charles F. Timmons and Jack M. Raisanen
    Citation: Acta Neuropathologica Communications 2016 4:55
  24. Low-grade gliomas (LGGs) account for about a third of all brain tumours in children. We conducted a detailed study of DNA methylation and gene expression to improve our understanding of the biology of pilocyti...

    Authors: Jennie N. Jeyapalan, Gabriel T. Doctor, Tania A. Jones, Samuel N. Alberman, Alexander Tep, Chirag M. Haria, Edward C. Schwalbe, Isabel C. F. Morley, Alfred A. Hill, Magdalena LeCain, Diego Ottaviani, Steven C. Clifford, Ibrahim Qaddoumi, Ruth G. Tatevossian, David W. Ellison and Denise Sheer
    Citation: Acta Neuropathologica Communications 2016 4:54
  25. Purkinje cell pathology is a common finding in a range of inherited and acquired cerebellar disorders, with the degree of Purkinje cell injury dependent on the underlying aetiology. Purkinje cells have an unpa...

    Authors: Kevin C. Kemp, Amelia J. Cook, Juliana Redondo, Kathreena M. Kurian, Neil J. Scolding and Alastair Wilkins
    Citation: Acta Neuropathologica Communications 2016 4:53

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:67

  26. Niemann-Pick type C (NPC) disease is a lysosomal storage disorder characterized by the occurrence of visceral and neurological symptoms. At present, the molecular mechanisms causing neurodegeneration in this d...

    Authors: A. Dardis, S. Zampieri, S. Canterini, K. L. Newell, C. Stuani, J. R. Murrell, B. Ghetti, M. T. Fiorenza, B. Bembi and E. Buratti
    Citation: Acta Neuropathologica Communications 2016 4:52
  27. Hexanucleotide repeat expansion in the C9orf72 gene is a leading cause of frontotemporal lobar degeneration (FTLD) with amyotrophic lateral sclerosis (ALS). Reduced expression of C9orf72 has been proposed as a...

    Authors: Peter M. Sullivan, Xiaolai Zhou, Adam M. Robins, Daniel H. Paushter, Dongsung Kim, Marcus B. Smolka and Fenghua Hu
    Citation: Acta Neuropathologica Communications 2016 4:51
  28. Pneumococcal meningitis is the most common and severe form of bacterial meningitis. Fatality rates are substantial, and long-term sequelae develop in about half of survivors. Here, we have performed a prospect...

    Authors: Mercedes Valls Serón, Bart Ferwerda, JooYeon Engelen-Lee, Madelijn Geldhoff, Valery Jaspers, Aeilko H. Zwinderman, Michael W. Tanck, Frank Baas, Arie van der Ende, Matthijs C. Brouwer and Diederik van de Beek
    Citation: Acta Neuropathologica Communications 2016 4:50
  29. Alzheimer’s disease (AD) is a progressive neurodegenerative disorder characterised by neuropathological deposits of amyloid plaques and neurofibrillary tangles comprised of β-amyloid and tau protein, respectiv...

    Authors: Dawn H. W. Lau, Marte Hogseth, Emma C. Phillips, Michael J. O’Neill, Amy M. Pooler, Wendy Noble and Diane P. Hanger
    Citation: Acta Neuropathologica Communications 2016 4:49
  30. Enlargement of a pre-existing intracranial aneurysm is a well-established risk factor of rupture. Excessive low wall shear stress concomitant with turbulent flow in the dome of an aneurysm may contribute to pr...

    Authors: Tomohiro Aoki, Kimiko Yamamoto, Miyuki Fukuda, Yuji Shimogonya, Shunichi Fukuda and Shuh Narumiya
    Citation: Acta Neuropathologica Communications 2016 4:48
  31. Dysregulation of RNA metabolism represents an important pathogenetic mechanism in both amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) due to the involvement of the DNA/RNA-binding protei...

    Authors: Elisa Onesto, Claudia Colombrita, Valentina Gumina, Maria Orietta Borghi, Sabrina Dusi, Alberto Doretti, Gigliola Fagiolari, Federica Invernizzi, Maurizio Moggio, Valeria Tiranti, Vincenzo Silani and Antonia Ratti
    Citation: Acta Neuropathologica Communications 2016 4:47
  32. Dorsal root ganglia (DRG) are highly vulnerable to frataxin deficiency in Friedreich ataxia (FA), an autosomal recessive disease due to pathogenic homozygous guanine-adenine-adenine trinucleotide repeat expans...

    Authors: Arnulf H. Koeppen, R. Liane Ramirez, Alyssa B. Becker and Joseph E. Mazurkiewicz
    Citation: Acta Neuropathologica Communications 2016 4:46
  33. Dermatomyositis (DM) can occur in both adults and juveniles with considerable clinical differences. The links between immune-mediated mechanisms and vasculopathy with respect to development of perifascicular p...

    Authors: Corinna Preuße, Yves Allenbach, Olaf Hoffmann, Hans-Hilmar Goebel, Debora Pehl, Josefine Radke, Alexandra Doeser, Udo Schneider, Rieke H.E. Alten, Tilmann Kallinich, Olivier Benveniste, Arpad von Moers, Benedikt Schoser, Ulrike Schara and Werner Stenzel
    Citation: Acta Neuropathologica Communications 2016 4:45
  34. Mutations of the human plectin gene (PLEC) on chromosome 8q24 cause autosomal recessive epidermolysis bullosa simplex with muscular dystrophy (EBS-MD). In the present study we analyzed the downstream effects of P...

    Authors: Lilli Winter, Matthias Türk, Patrick N. Harter, Michel Mittelbronn, Cornelia Kornblum, Fiona Norwood, Heinz Jungbluth, Christian T. Thiel, Ursula Schlötzer-Schrehardt and Rolf Schröder
    Citation: Acta Neuropathologica Communications 2016 4:44
  35. Approximately 20 % of familial Amyotrophic Lateral Sclerosis (ALS) is caused by mutations in superoxide dismutase (SOD1), which leads to misfolding of the SOD1 protein, resulting in a toxic gain of function. Seve...

    Authors: Sarah Pickles, Sabrina Semmler, Helen R. Broom, Laurie Destroismaisons, Laurine Legroux, Nathalie Arbour, Elizabeth Meiering, Neil R. Cashman and Christine Vande Velde
    Citation: Acta Neuropathologica Communications 2016 4:43
  36. There is growing evidence that defective DNA repair in neurons with accumulation of DNA lesions and loss of genome integrity underlies aging and many neurodegenerative disorders. An important challenge is to u...

    Authors: Jorge Mata-Garrido, Iñigo Casafont, Olga Tapia, Maria T. Berciano and Miguel Lafarga
    Citation: Acta Neuropathologica Communications 2016 4:41
  37. Primary central nervous system lymphoma (PCNSL) is an aggressive variant of diffuse large B-cell lymphoma (DLBCL) confined to the CNS. TP53 mutations (MUT-TP53) were investigated in the context of MIR34A/B/C- and...

    Authors: Helga D. Munch-Petersen, Fazila Asmar, Konstantinos Dimopoulos, Aušrinė Areškevičiūtė, Peter Brown, Mia Seremet Girkov, Anja Pedersen, Lene D. Sjö, Steffen Heegaard, Helle Broholm, Lasse S. Kristensen, Elisabeth Ralfkiaer and Kirsten Grønbæk
    Citation: Acta Neuropathologica Communications 2016 4:40
  38. Parkinson’s disease (PD) is the most common neurodegenerative movement disorder, yet disease-modifying treatments do not currently exist. Rho-associated protein kinase (ROCK) was recently described as a novel ...

    Authors: Lars Tatenhorst, Katrin Eckermann, Vivian Dambeck, Luis Fonseca-Ornelas, Hagen Walle, Tomás Lopes da Fonseca, Jan C. Koch, Stefan Becker, Lars Tönges, Mathias Bähr, Tiago F. Outeiro, Markus Zweckstetter and Paul Lingor
    Citation: Acta Neuropathologica Communications 2016 4:39
  39. Neurological diseases associated with neuronal death are also accompanied by axonal denervation of connected brain regions. In these areas, denervation leads to a decrease in afferent drive, which may in turn ...

    Authors: David Verbich, Denise Becker, Andreas Vlachos, Peter Mundel, Thomas Deller and R. Anne McKinney
    Citation: Acta Neuropathologica Communications 2016 4:38
  40. A non-coding hexanucleotide repeat expansion (HRE) in C9orf72 is a common cause of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) acting through a loss of function mechanism due to haploins...

    Authors: Patrizia Rizzu, Cornelis Blauwendraat, Sasja Heetveld, Emily M. Lynes, Melissa Castillo-Lizardo, Ashutosh Dhingra, Elwira Pyz, Markus Hobert, Matthis Synofzik, Javier Simón-Sánchez, Margherita Francescatto and Peter Heutink
    Citation: Acta Neuropathologica Communications 2016 4:37
  41. Choreoathetoid involuntary movements are rarely reported in patients with frontotemporal lobar degeneration (FTLD), suggesting their exclusion as a supportive feature in clinical diagnostic criteria for FTLD. ...

    Authors: Ito Kawakami, Zen Kobayashi, Tetsuaki Arai, Osamu Yokota, Takashi Nonaka, Naoya Aoki, Kazuhiro Niizato, Kenichi Oshima, Shinji Higashi, Omi Katsuse, Masato Hosokawa, Masato Hasegawa and Haruhiko Akiyama
    Citation: Acta Neuropathologica Communications 2016 4:36
  42. The observation showing that Lewy type synucleinopathy (LTS), the pathological hallmark of Parkinson’s disease (PD), is found in the gut of almost all PD subjects led to a substantial amount of research to dev...

    Authors: Anne-Gaëlle Corbillé, Franck Letournel, Jeffrey H. Kordower, John Lee, Elisheva Shanes, Michel Neunlist, David G. Munoz, Pascal Derkinderen and Thomas G. Beach
    Citation: Acta Neuropathologica Communications 2016 4:35
  43. Alterations in calcium homeostasis are widely reported to contribute to synaptic degeneration and neuronal loss in Alzheimer’s disease. Elevated cytosolic calcium concentrations lead to activation of the calci...

    Authors: Ksenia Kurbatskaya, Emma C. Phillips, Cara L. Croft, Giacomo Dentoni, Martina M. Hughes, Matthew A. Wade, Safa Al-Sarraj, Claire Troakes, Michael J. O’Neill, Beatriz G. Perez-Nievas, Diane P. Hanger and Wendy Noble
    Citation: Acta Neuropathologica Communications 2016 4:34
  44. It has been suggested that patients with motor neurone disease (MND) and those with MND combined with behavioural variant frontotemporal dementia (bvFTD) (ie FTD + MND) or with FTD alone might exist on a conti...

    Authors: Roya Behrouzi, Xiawei Liu, Dongyue Wu, Andrew C. Robinson, Sayuri Tanaguchi-Watanabe, Sara Rollinson, Jing Shi, Jinzhou Tian, Hisham H. M. Hamdalla, John Ealing, Anna Richardson, Matthew Jones, Stuart Pickering-Brown, Yvonne S. Davidson, Michael J. Strong, Masato Hasegawa…
    Citation: Acta Neuropathologica Communications 2016 4:33
  45. The phenotypic spectrum among girls with heterozygous mutations in the X-linked intellectual disability (XLID) gene CASK (calcium/calmodulin-dependent serine protein kinase) includes postnatal microcephaly, ponto...

    Authors: Sarika Srivastava, Ryan McMillan, Jeffery Willis, Helen Clark, Vrushali Chavan, Chen Liang, Haiyan Zhang, Matthew Hulver and Konark Mukherjee
    Citation: Acta Neuropathologica Communications 2016 4:30
  46. The transmembrane protein assembly γ-secretase is a key protease in regulated intramembrane processing (RIP) of around 100 type-1 transmembrane proteins. Importantly, it has a pathological role in Alzheimer di...

    Authors: Sophia Schedin-Weiss, Ina Caesar, Bengt Winblad, Hans Blom and Lars O. Tjernberg
    Citation: Acta Neuropathologica Communications 2016 4:29
  47. A hallmark of several major neurological diseases is neuronal cell death. In addition to this primary pathology, secondary injury is seen in connected brain regions in which neurons not directly affected by th...

    Authors: Laurent M. Willems, Nadine Zahn, Nerea Ferreirós, Klaus Scholich, Nicola Maggio, Thomas Deller and Andreas Vlachos
    Citation: Acta Neuropathologica Communications 2016 4:28

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    Journal Impact Factor: 6.2
    5-year Journal Impact Factor: 6.8
    Source Normalized Impact per Paper (SNIP): 1.344
    SCImago Journal Rank (SJR): 2.580

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    Submission to first editorial decision (median days): 3
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