Acta Neuropathologica Communications

ANC: Neuroscience of Disease

Featured video: Another mechanism for the initiation and progression of Parkinson's disease

A research team in the USA and China identifies a potential disease mechanism in the initiation and development of Parkinson’s disease.

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Content type: Research
| 24 April 2018

Genetic deletion of muscle RANK or selective inhibition of RANKL is not as effective as full-length OPG-fc in mitigating muscular dystrophy

Authors: Sébastien S. Dufresne, Antoine Boulanger-Piette, Sabrina Bossé, Anteneh Argaw, Dounia Hamoudi, Laetitia Marcadet, Daniel Gamu, Val A. Fajardo, Hideo Yagita, Josef M. Penninger, A. Russell Tupling and Jérôme Frenette
Content type: Research
| 24 April 2018

Prion infectivity is encoded exclusively within the structure of proteinase K-resistant fragments of synthetically generated recombinant PrP^Sc

Authors: Fei Wang, Xinhe Wang, Romany Abskharon and Jiyan Ma
Content type: Research
| 18 April 2018

Potent prion-like behaviors of pathogenic α-synuclein and evaluation of inactivation methods

Authors: Airi Tarutani, Tetsuaki Arai, Shigeo Murayama, Shin-ichi Hisanaga and Masato Hasegawa
Content type: Research
| 13 April 2018

Phosphorylated TDP-43 (pTDP-43) aggregates in the axial skeletal muscle of patients with sporadic and familial amyotrophic lateral sclerosis

Authors: Matthew D. Cykowski, Suzanne Z. Powell, Joan W. Appel, Anithachristy S. Arumanayagam, Andreana L. Rivera and Stanley H. Appel
Content type: Research
| 11 April 2018

Zidovudine ameliorates pathology in the mouse model of Duchenne muscular dystrophy via P2RX7 purinoceptor antagonism

Authors: Rasha Al-Khalidi, Chiara Panicucci, Paul Cox, Natalia Chira, Justyna Róg, Christopher N. J. Young, Rhiannon E. McGeehan, Kameshwari Ambati, Jayakrishna Ambati, Krzysztof Zabłocki, Elisabetta Gazzerro, Stephen Arkle, Claudio Bruno and Dariusz C. Górecki

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Content type: Research
| 2 August 2013

Myelin alters the inflammatory phenotype of macrophages by activating PPARs

Authors: Jeroen FJ Bogie, Winde Jorissen, Jo Mailleux, Philip G Nijland, Noam Zelcer, Tim Vanmierlo, Jack Van Horssen, Piet Stinissen, Niels Hellings and Jerome JA Hendriks
Content type: Research
| 9 May 2013

Do brain T2/FLAIR white matter hyperintensities correspond to myelin loss in normal aging? A radiologic-neuropathologic correlation study

Authors: Sven Haller, Enikö Kövari, François R Herrmann, Victor Cuvinciuc, Ann-Marie Tomm, Gilbert B Zulian, Karl-Olof Lovblad, Panteleimon Giannakopoulos and Constantin Bouras
Content type: Research
| 15 July 2014

Possible involvement of lysosomal dysfunction in pathological changes of the brain in aged progranulin-deficient mice

Authors: Yoshinori Tanaka, James K Chambers, Takashi Matsuwaki, Keitaro Yamanouchi and Masugi Nishihara
Content type: Case report
| 2 August 2013

Neuropathology of brain and spinal malformations in a case of monosomy 1p36

Authors: Naoko Shiba, Ray AM Daza, Lisa G Shaffer, A James Barkovich, William B Dobyns and Robert F Hevner
Content type: Review
| 8 May 2013

The prion hypothesis in Parkinson's disease: Braak to the future

Authors: Naomi P Visanji, Patricia L Brooks, Lili-Naz Hazrati and Anthony E Lang

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The characterization of extracellular vesicles from packed red blood cells

Featured article: Transmission of α-synuclein-containing erythrocyte-derived extracellular vesicles across the blood-brain barrier via adsorptive mediated transcytosis: another mechanism for initiation and progression of Parkinson’s disease?

Professor Zhang et al investigate the mechanism of extracellular vesicle transmission across the blood-brain barrier as it relates to Parkinson's Disease.

Featured article: Novel compound PBT434 prevents iron mediated neurodegeneration and alpha-synuclein toxicity in multiple models of Parkinson’s disease

Dr. Finkelstein et al report the preclinical characterization of PBT434, a novel quinazolinone compound bearing a moderate affinity metal-binding motif, which is in development for Parkinsonian conditions.

EUROTAU: Reflections on the Status Quo

A series of articles from the first EUROTAU meeting.

EUROTAU is an innovative, collaborative research initiative established to tackle unanswered research questions and the mounting challenges posed by Tauopathies.

Editors-in-Chief

Werner Paulus, University of Münster, Germany
Johannes Attems, Newcastle University, UK

Aims and scope

Acta Neuropathologica Communications (ANC) focuses on the pathogenesis of neurological disease using experimental or human tissues, molecular, cellular, biological and morphological techniques.

Why publish with ANC?

No major revisions: only minor revisions are requested that can be performed (and have to be performed) within 3 weeks
Fast, thorough peer review: group of editors selected on the basis of their insightful, dedicated and prompt service
Rapid publication: publication within 2 months of submission
Inclusion in PubMed, PubMed Central and MEDLINE
Open access: enabling it to be read by a much larger set of readers, increasing its visibility
Competitive APC: flat article-processing charge, with no page limits, color figure charges or submission fees

Frequently Asked Questions

Our FAQs about the journal are answered

Our commitment to excellent author service

ANC is committed to rapid publication. We aim to publish all research articles within 2 months of submission, subject to authors responding to requests within the required timeframe. If the publication of your article exceeds 2 months from submission, you may be entitled to a full waiver or discount of the article-processing charge on your next submission to the journal. This policy excludes manuscripts delayed for reasons outside of the Publisher's control, such as delays resulting from ethics-related issues or queries concerning your manuscript. All claims will be considered on a case-by-case basis. For further information or to make a claim, please contact editorial@actaneurocomms.org.

Sister journal

ANC is sister journal to Acta Neuropathologica, a journal dedicated to the pathology and mechanisms of neurological disease.

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Editors' quotes

"Acta Neuropathologica Communications is an open access, peer-reviewed online journal. Like our sister journal Acta Neuropathologica we focus on pathology and mechanisms of neurological disease. We publish high quality articles that do not require major manuscript revisions. We offer very fast turnaround times, publication of papers within two months following submission, enthusiastic editors, and a competitive article-processing charge. Try it out!"

Werner Paulus, co-Editor-in-Chief, ANC

"Acta Neuropathologica Communications is a journal for researchers working on human neurological disease and has a focus on neuropathology. However, since we believe that an interdisciplinary approach is highly valuable to advance the field, our journal is not restricted to classical neuropathological articles. Therefore, we also welcome high-quality submissions from other disciplines as long as they do ultimately aim to advance our understanding of human neurological disease. We try our best to help you with the submission process and in case of any questions do not hesitate to contact the Editors-in-Chief directly."

Johannes Attems, co-Editor-in-Chief, ANC

Acta Neuropathologica Communications

ANC: Neuroscience of Disease

Featured video: Another mechanism for the initiation and progression of Parkinson's disease

Articles

Genetic deletion of muscle RANK or selective inhibition of RANKL is not as effective as full-length OPG-fc in mitigating muscular dystrophy

Prion infectivity is encoded exclusively within the structure of proteinase K-resistant fragments of synthetically generated recombinant PrP^Sc

Potent prion-like behaviors of pathogenic α-synuclein and evaluation of inactivation methods

Phosphorylated TDP-43 (pTDP-43) aggregates in the axial skeletal muscle of patients with sporadic and familial amyotrophic lateral sclerosis

Zidovudine ameliorates pathology in the mouse model of Duchenne muscular dystrophy via P2RX7 purinoceptor antagonism

Myelin alters the inflammatory phenotype of macrophages by activating PPARs

Do brain T2/FLAIR white matter hyperintensities correspond to myelin loss in normal aging? A radiologic-neuropathologic correlation study

Possible involvement of lysosomal dysfunction in pathological changes of the brain in aged progranulin-deficient mice

Neuropathology of brain and spinal malformations in a case of monosomy 1p36

The prion hypothesis in Parkinson's disease: Braak to the future

Featured article: Transmission of α-synuclein-containing erythrocyte-derived extracellular vesicles across the blood-brain barrier via adsorptive mediated transcytosis: another mechanism for initiation and progression of Parkinson’s disease?

Featured article: Novel compound PBT434 prevents iron mediated neurodegeneration and alpha-synuclein toxicity in multiple models of Parkinson’s disease

EUROTAU: Reflections on the Status Quo

Editors-in-Chief

Aims and scope

Why publish with ANC?

Frequently Asked Questions

Our commitment to excellent author service

Sister journal

Latest Tweets

Editors' quotes

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2016 Journal Metrics

Acta Neuropathologica Communications

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