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Correction to: Hsp90 co‑chaperones, FKBP52 and Aha1, promote tau pathogenesis in aged wild‑type mice

The Original Article was published on 08 April 2021

Correction to: Criado-Marrero et al. acta neuropathol commun (2021) 9, 65 https://doi.org/10.1186/s40478-021-01159-w

After publication of this article [1], it is noticed this article contained an error: Figure 4 was incorrect.

The correct Fig. 4 has been provided in this Correction.

Fig. 4
figure 4

Examples of tau accumulation in the hippocampus of aged wild‑type mice following overexpression of Aha1 or FKBP52. High magnification images (×100) were obtained from the hippocampus of a representative animal in the group with the highest tau accumulation. Representative images of tau species and their respective groups are the following: (a) total tau (Dako; AAV9‑Aha1), (b) pT231 tau (AAV9‑Aha1), (c) AT8 tau (pS202/T205; AAV9‑FKBP52), (d) pS396 tau (AAV9‑FKBP52), (e) T22 (AAV9‑Aha1), and (f) Gallyas‑silver (AAV9‑FKBP52). Scale bar represents 10 µm

The original article has been updated.

Reference

  1. Criado-Marrero M, Gebru NT, Blazier DM et al (2021) Hsp90 co-chaperones, FKBP52 and Aha1, promote tau pathogenesis in aged wild-type mice. Acta Neuropathol Commun 9:65. https://doi.org/10.1186/s40478-021-01159-w

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Correspondence to Laura J. Blair.

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Criado-Marrero, M., Gebru, N.T., Blazier, D.M. et al. Correction to: Hsp90 co‑chaperones, FKBP52 and Aha1, promote tau pathogenesis in aged wild‑type mice. acta neuropathol commun 9, 85 (2021). https://doi.org/10.1186/s40478-021-01188-5

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  • DOI: https://doi.org/10.1186/s40478-021-01188-5