Fig. 6From: BDNF-dependent modulation of axonal transport is selectively impaired in ALSTrkB and p75NTR levels are comparable at the NMJ between tibialis anterior (TA) and soleus muscles, and are not altered in disease. NMJ immunostainings of A (i) TrkB and B (i) p75NTR receptors at the pre- and post-synaptic compartments as well as in terminal Schwann cells as identified by ii) Syn/TUJ1, iii) ⍺-BTX, and iv) S100 staining, respectively. The boxed area in A-Bi-v ‘ is enlarged in A-Bi-v“. Scale bars in v' = 10 µm, scale bars in v" = 5 µm. Immunostaining quantifications of: C TrkB (genotype p = 0.868, muscle p = 0.109, interaction p = 0.781); and D p75.NTR (genotype p = 0.091, muscle p = 0.108, interaction p = 0.603) (n = 6). Data were compared by two-way ANOVA and Holm-Šídák's multiple comparisons tests. Means ± SEM are plotted for all graphs. Black (P73) and grey (P94) circle borders indicate age-matched mice. See also Additional file 2: Fig. S6Back to article page