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  1. Like uveal melanomas, primary leptomeningeal melanocytic neoplasms (LMNs) frequently carry GNAQ and GNA11 mutations. However, it is currently unknown whether these LMNs harbor mutations in BAP1, SF3B1 and/or EIF1...

    Authors: Heidi V. N. Küsters-Vandevelde, David Creytens, Adriana C. H. van Engen-van Grunsven, Marcel Jeunink, Veronique Winnepenninckx, Patricia J. T. A. Groenen, Benno Küsters, Pieter Wesseling, Willeke A. M. Blokx and Clemens F. M. Prinsen
    Citation: Acta Neuropathologica Communications 2016 4:5
  2. Glioblastoma with oligodendroglioma component (GBM-O) was recognized as a histologic pattern of glioblastoma (GBM) by the World Health Organization (WHO) in 2007 and is distinguished by the presence of oligode...

    Authors: Benjamin H. Hinrichs, Scott Newman, Christina L. Appin, William Dunn, Lee Cooper, Rini Pauly, Jeanne Kowalski, Michael R. Rossi and Daniel J. Brat
    Citation: Acta Neuropathologica Communications 2016 4:4
  3. Amyotrophic lateral sclerosis (ALS) is a motor neuron disease with a gender bias towards major prevalence in male individuals. Several data suggest the involvement of oxidative stress and mitochondrial dysfunc...

    Authors: Daniel Cacabelos, Omar Ramírez-Núñez, Ana Belén Granado-Serrano, Pascual Torres, Victòria Ayala, Victoria Moiseeva, Mònica Povedano, Isidre Ferrer, Reinald Pamplona, Manuel Portero-Otin and Jordi Boada
    Citation: Acta Neuropathologica Communications 2016 4:3
  4. Diffuse intrinsic pontine glioma (DIPG) and midline high-grade glioma (mHGG) are lethal childhood brain tumors. Spatial genomic heterogeneity has been well-described in adult HGG but has not been comprehensive...

    Authors: Lindsey M. Hoffman, Mariko DeWire, Scott Ryall, Pawel Buczkowicz, James Leach, Lili Miles, Arun Ramani, Michael Brudno, Shiva Senthil Kumar, Rachid Drissi, Phillip Dexheimer, Ralph Salloum, Lionel Chow, Trent Hummel, Charles Stevenson, Q. Richard Lu…
    Citation: Acta Neuropathologica Communications 2016 4:1

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:13

  5. CADASIL (Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy) is a hereditary small vessel disease caused by mutations in the NOTCH3 gene, leading to toxic NOTCH3 protein ac...

    Authors: Julie W. Rutten, Roselin R. Klever, Ingrid M. Hegeman, Dana S. Poole, Hans G. Dauwerse, Ludo A. M. Broos, Cor Breukel, Annemieke M. Aartsma-Rus, J. Sjef Verbeek, Louise van der Weerd, Sjoerd G. van Duinen, Arn M. J. M. van den Maagdenberg and Saskia A. J. Lesnik Oberstein
    Citation: Acta Neuropathologica Communications 2015 3:89
  6. Recent advances in molecular diagnostics allow diffuse gliomas to be classified based on their genetic changes into distinct prognostic subtypes. However, a systematic analysis of all molecular markers has thu...

    Authors: Kaspar Draaisma, Maarten M. J. Wijnenga, Bas Weenink, Ya Gao, Marcel Smid, P. Robe, Martin J. van den Bent and Pim J. French
    Citation: Acta Neuropathologica Communications 2015 3:88
  7. The important protective role of small heat-shock proteins (HSPs) in regulating cellular survival and migration, counteracting protein aggregation, preventing apoptosis, and regulating inflammation in the cent...

    Authors: Laura A. N. Peferoen, Wouter H. Gerritsen, Marjolein Breur, Kimberley M. D. Ummenthum, Regina M. B. Peferoen-Baert, Paul van der Valk, Johannes M. van Noort and Sandra Amor
    Citation: Acta Neuropathologica Communications 2015 3:87
  8. Pilocytic astrocytomas are slow-growing tumors that usually occur in the cerebellum or in the midline along the hypothalamic/optic pathways. The most common genetic alterations in pilocytic astrocytomas activa...

    Authors: Tania A. Jones, Jennie N. Jeyapalan, Tim Forshew, Ruth G. Tatevossian, Andrew R. J. Lawson, Sheena N. Patel, Gabriel T. Doctor, Muhammad A. Mumin, Simon R. Picker, Kim P. Phipps, Antony Michalski, Thomas S. Jacques and Denise Sheer
    Citation: Acta Neuropathologica Communications 2015 3:86
  9. Papillary Glioneuronal Tumor (PGNT) is a grade I tumor which was classified as a separate entity in the World Health Organization Classification of the Central Nervous System 2007 in the group of mixed glioneu...

    Authors: Melanie Pages, Ludovic Lacroix, Arnault Tauziede-Espariat, David Castel, Estelle Daudigeos-Dubus, Vita Ridola, Sophie Gilles, Frederic Fina, Felipe Andreiuolo, Marc Polivka, Emmanuele Lechapt-Zalcman, Stephanie Puget, Nathalie Boddaert, Xiao-qiong Liu, Julia A. Bridge, Jacques Grill…
    Citation: Acta Neuropathologica Communications 2015 3:85
  10. The early clinical trials using fetal ventral mesencephalic (VM) allografts in Parkinson’s disease (PD) patients have shown efficacy (albeit not in all cases) and have paved the way for further development of ...

    Authors: Anna Tamburrino, Madeline J. Churchill, Oi W. Wan, Yolanda Colino-Sanguino, Rossana Ippolito, Sofie Bergstrand, Daniel A. Wolf, Niculin J. Herz, Michelle D. Sconce, Anders Björklund, Charles K. Meshul and Mickael Decressac
    Citation: Acta Neuropathologica Communications 2015 3:84
  11. Alzheimer’s disease (AD) is the most dominant neurodegenerative disorder that causes dementia, and no effective treatments are available. To study its pathogenesis and develop therapeutics, animal models repre...

    Authors: James K. Chambers, Takahiko Tokuda, Kazuyuki Uchida, Ryotaro Ishii, Harutsugu Tatebe, Erika Takahashi, Takami Tomiyama, Yumi Une and Hiroyuki Nakayama
    Citation: Acta Neuropathologica Communications 2015 3:78
  12. Authors: Christopher M. Henstridge, Rosemary J. Jackson, JeeSoo M. Kim, Abigail G. Herrmann, Ann K. Wright, Sarah E. Harris, Mark E. Bastin, John M. Starr, Joanna Wardlaw, Thomas H. Gillingwater, Colin Smith, Chris-Anne McKenzie, Simon R. Cox, Ian J. Deary and Tara L. Spires-Jones
    Citation: Acta Neuropathologica Communications 2015 3:83

    The original article was published in Acta Neuropathologica Communications 2015 3:53

  13. Neuromyelitis optica (NMO), an autoimmune astrocytopathic disease associated with anti-aquaporin-4 (AQP4) antibody, is characterized by extensive necrotic lesions preferentially involving the optic nerves and ...

    Authors: Kazuhiro Kurosawa, Tatsuro Misu, Yoshiki Takai, Douglas Kazutoshi Sato, Toshiyuki Takahashi, Yoichiro Abe, Hiroko Iwanari, Ryo Ogawa, Ichiro Nakashima, Kazuo Fujihara, Takao Hamakubo, Masato Yasui and Masashi Aoki
    Citation: Acta Neuropathologica Communications 2015 3:82
  14. Smooth muscle cell contraction is an essential function of arteries and relies on the integrity of the actin-myosin apparatus. The tissue-specific α2-smooth muscle actin, encoded by ACTA2, is predominantly expres...

    Authors: Maria-Magdalena Georgescu, Marco da Cunha Pinho, Timothy E. Richardson, Jose Torrealba, L. Maximilian Buja, Dianna M. Milewicz, Jack M. Raisanen and Dennis K. Burns
    Citation: Acta Neuropathologica Communications 2015 3:81
  15. Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system. Immunological studies suggest that it is a T-cell mediated autoimmune disease, although an MS-specific tar...

    Authors: Romana Höftberger, Marianne Leisser, Jan Bauer and Hans Lassmann
    Citation: Acta Neuropathologica Communications 2015 3:80
  16. Demyelinated axons in multiple sclerosis (MS) lesions have an increased energy demand in order to maintain conduction. However, oxidative stress-induced mitochondrial dysfunction likely alters glucose metaboli...

    Authors: Philip G. Nijland, Remco J. Molenaar, Susanne M. A. van der Pol, Paul van der Valk, Cornelis J. F. van Noorden, Helga E. de Vries and Jack van Horssen
    Citation: Acta Neuropathologica Communications 2015 3:79
  17. Depletion of cholinergic neurons within the nucleus basalis of Meynert (nbM) is thought to contribute to the development of cognitive impairments in both Alzheimer’s disease (AD) and Lewy body disorders (LBD)....

    Authors: Athanasios Alexandris, Alan King Lun Liu, Raymond Chuen-Chung Chang, Ronald K. B. Pearce and Steve M. Gentleman
    Citation: Acta Neuropathologica Communications 2015 3:77
  18. Parkinson’s disease (PD) is characterized by the accumulation of abnormal α-synuclein in selected regions of the brain following a gradient of severity with disease progression. Whether this is accompanied by ...

    Authors: Paula Garcia-Esparcia, Karina Hernández-Ortega, Anusha Koneti, Laura Gil, Raul Delgado-Morales, Ester Castaño, Margarita Carmona and Isidre Ferrer
    Citation: Acta Neuropathologica Communications 2015 3:76
  19. Parkinson’s disease (PD) and multiple system atrophy (MSA) are neurodegenerative diseases that are characterized by the intracellular accumulation of alpha-synuclein containing aggregates. Recent increasing ev...

    Authors: Maria E. Bernis, Julius T. Babila, Sara Breid, Katharina Annick Wüsten, Ullrich Wüllner and Gültekin Tamgüney
    Citation: Acta Neuropathologica Communications 2015 3:75
  20. Tamoxifen (TAM) is an important cancer therapeutic and an experimental tool for effecting genetic recombination using the inducible Cre-Lox technique. Despite its widespread use in the clinic and laboratory, w...

    Authors: Franziska Denk, Leanne M. Ramer, Erin L. K. S. Erskine, Mohammed A. Nassar, Yury Bogdanov, Massimo Signore, John N. Wood, Stephen B. McMahon and Matt S. Ramer
    Citation: Acta Neuropathologica Communications 2015 3:74
  21. Autosomal dominant adult-onset neuronal ceroid lipofuscinosis (AD-ANCL) is a multisystem disease caused by mutations in the DNAJC5 gene. DNAJC5 encodes Cysteine String Protein-alpha (CSPα), a putative synaptic pr...

    Authors: Bruno A. Benitez, Nigel J. Cairns, Robert E. Schmidt, John C. Morris, Joanne B. Norton, Carlos Cruchaga and Mark S. Sands
    Citation: Acta Neuropathologica Communications 2015 3:73
  22. Transgenic overexpression of amyloid precursor protein (APP) genes that are either entirely human in sequence or have humanized Aβ sequences can produce Alzheimer-type amyloidosis in mice, provided the transge...

    Authors: Guilian Xu, Yong Ran, Susan E. Fromholt, Chunhua Fu, Anthony T. Yachnis, Todd E. Golde and David R. Borchelt
    Citation: Acta Neuropathologica Communications 2015 3:72
  23. In Alzheimer’s disease, accumulation and pathological aggregation of amyloid β-peptide is accompanied by the induction of complex immune responses, which have been attributed both beneficial and detrimental pr...

    Authors: Claudia Späni, Tobias Suter, Rebecca Derungs, Maria Teresa Ferretti, Tobias Welt, Fabian Wirth, Christoph Gericke, Roger M. Nitsch and Luka Kulic
    Citation: Acta Neuropathologica Communications 2015 3:71
  24. Amyloid β (Aβ) accumulates in the extracellular space as diffuse and neuritic plaques in Alzheimer’s disease (AD). Aβ also deposits on the walls of arterioles as cerebral amyloid angiopathy (CAA) in most cases...

    Authors: Fan Liao, Tony J. Zhang, Hong Jiang, Katheryn B. Lefton, Grace O. Robinson, Robert Vassar, Patrick M. Sullivan and David M. Holtzman
    Citation: Acta Neuropathologica Communications 2015 3:70
  25. TAR DNA-binding protein 43 (TDP-43) inclusions are pathological hallmarks of patients with frontotemporal lobar degeneration (FTLD) and amyotrophic lateral sclerosis (ALS). Loss of TDP-43 in zebrafish engender...

    Authors: Jonathan Janssens, Stéphanie Philtjens, Gernot Kleinberger, Sara Van Mossevelde, Julie van der Zee, Rita Cacace, Sebastiaan Engelborghs, Anne Sieben, Julia Banzhaf-Strathmann, Lubina Dillen, Céline Merlin, Ivy Cuijt, Caroline Robberecht, Bettina Schmid, Patrick Santens, Adrian Ivanoiu…
    Citation: Acta Neuropathologica Communications 2015 3:68
  26. Septic encephalopathy is associated with rapid deterioration of cortical functions. Using magnetic resonance imaging (MRI) we detected functional abnormalities in the hippocampal formation of patients with sep...

    Authors: Aleksandar R. Zivkovic, Oliver Sedlaczek, Rebecca von Haken, Karsten Schmidt, Thorsten Brenner, Markus A. Weigand, Hilmar Bading, C. Peter Bengtson and Stefan Hofer
    Citation: Acta Neuropathologica Communications 2015 3:67
  27. The A673T mutation in the amyloid precursor protein (APP) protects against Alzheimer’s disease by reducing β-amyloid protein (Aβ) production. This mutation reduced the release of the soluble APP fragment (sAPP...

    Authors: Asuka Kokawa, Seiko Ishihara, Hitomi Fujiwara, Mika Nobuhara, Minori Iwata, Yasuo Ihara and Satoru Funamoto
    Citation: Acta Neuropathologica Communications 2015 3:66
  28. Muscle stem cells termed satellite cells are essential for muscle regeneration. A central question in many neuromuscular disorders is why satellite cells are unable to prevent progressive muscle wasting. We ha...

    Authors: Gerben J. Schaaf, Tom JM van Gestel, Esther Brusse, Robert M. Verdijk, Irenaeus FM de Coo, Pieter A. van Doorn, Ans T. van der Ploeg and WWM Pim Pijnappel
    Citation: Acta Neuropathologica Communications 2015 3:65
  29. Autism is diagnosed in numerous genetic and genomic developmental disorders associated with an overlap in high-risk genes and loci that underlie intellectual disability (ID) and epilepsy. The aim of this stere...

    Authors: Jerzy Wegiel, Michael Flory, N. Carolyn Schanen, Edwin H. Cook, Krzysztof Nowicki, Izabela Kuchna, Humi Imaki, Shuang Yong Ma, Jarek Wegiel, Eric London, Manuel F. Casanova, Thomas Wisniewski and W. Ted Brown
    Citation: Acta Neuropathologica Communications 2015 3:63
  30. Mutations in the FUS gene have been shown to be a rare cause of amyotrophic lateral sclerosis (ALS-FUS) and whilst well documented clinically and genetically there have been relatively few neuropathological studi...

    Authors: Andrew King, Claire Troakes, Bradley Smith, Matthew Nolan, Olimpia Curran, Caroline Vance, Christopher E. Shaw and Safa Al-Sarraj
    Citation: Acta Neuropathologica Communications 2015 3:62
  31. Ischemic stroke, a major cause of mortality, is frequently accompanied by life-threatening cerebral edema. Aquaporin-4 (Aqp4), an astrocytic transmembrane water channel, is an important molecular contributor t...

    Authors: Jesse A. Stokum, Rupal I. Mehta, Svetlana Ivanova, Edward Yu, Volodymyr Gerzanich and J. Marc Simard
    Citation: Acta Neuropathologica Communications 2015 3:61
  32. Cerebral white matter lesions (WML), visualized as white matter hyperintensities (WMH) on T2-weighted MRI, encompass structural damage and loss of integrity of the cerebral white matter (WM) and are commonly a...

    Authors: Kirsty E. McAleese, Michael Firbank, Madhurima Dey, Sean J. Colloby, Lauren Walker, Mary Johnson, Joshua R. Beverley, John Paul Taylor, Alan J. Thomas, John T. O’Brien and Johannes Attems
    Citation: Acta Neuropathologica Communications 2015 3:60
  33. A majority of familial frontotemporal lobar dementia and amyotrophic lateral sclerosis cases are associated with a large repeat expansion in a non-coding region of the C9ORF72 gene. Currently, little is known abo...

    Authors: Rachel A K Atkinson, Carmen M. Fernandez-Martos, Julie D. Atkin, James C. Vickers and Anna E. King
    Citation: Acta Neuropathologica Communications 2015 3:59

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:2

  34. The application of high-throughput genomic approaches has revealed 24 novel risk loci for Alzheimer’s disease (AD). We recently reported that the bridging integrator 1 (BIN1) risk gene is linked to Tau pathology.

    Authors: Yoann Sottejeau, Alexis Bretteville, François-Xavier Cantrelle, Nicolas Malmanche, Florie Demiaute, Tiago Mendes, Charlotte Delay, Harmony Alves Dos Alves, Amandine Flaig, Peter Davies, Pierre Dourlen, Bart Dermaut, Jocelyn Laporte, Philippe Amouyel, Guy Lippens, Julien Chapuis…
    Citation: Acta Neuropathologica Communications 2015 3:58
  35. Lewy body–related α-synucleinopathy (LBAS, the abnormal accumulation of pathologic α-synuclein) is found in the central and peripheral nervous systems, including the spinal cord, dorsal root ganglia, and sympa...

    Authors: Hiroyuki Sumikura, Masaki Takao, Hiroyuki Hatsuta, Shinji Ito, Yuta Nakano, Akiko Uchino, Akane Nogami, Yuko Saito, Hideki Mochizuki and Shigeo Murayama
    Citation: Acta Neuropathologica Communications 2015 3:57
  36. Several studies have indicated that autoimmune and neuroinflammatory processes contribute to the neurodegeneration of retinal ganglion cells in human glaucoma patients and in animal models. To test the involve...

    Authors: Oliver W. Gramlich, Qiong J. Ding, Wei Zhu, Amy Cook, Michael G. Anderson and Markus H. Kuehn
    Citation: Acta Neuropathologica Communications 2015 3:56
  37. The term motor neuron disease encompasses a spectrum of disorders in which motor neurons are the lost. Importantly, while some motor neurons are lost early in disease and others remain intact at disease end-st...

    Authors: Lyndsay M. Murray, Ariane Beauvais, Sabrina Gibeault, Natalie L. Courtney and Rashmi Kothary
    Citation: Acta Neuropathologica Communications 2015 3:55
  38. Substantial data has shown that the lectican group of chondroitin sulfate proteoglycans are involved in inhibition of axonal plasticity in response to injury in the central nervous system. Increasing evidence ...

    Authors: Matthew D. Howell, Lauren A. Bailey, Michael A. Cozart, Brenda M. Gannon and Paul E. Gottschall
    Citation: Acta Neuropathologica Communications 2015 3:54
  39. Non-pathological, age-related cognitive decline varies markedly between individuals and places significant financial and emotional strain on people, their families and society as a whole. Understanding the dif...

    Authors: Christopher M. Henstridge, Rosemary J. Jackson, JeeSoo M. Kim, Abigail G. Herrmann, Ann K. Wright, Sarah E. Harris, Mark E. Bastin, John M. Starr, Joanna Wardlaw, Thomas H. Gillingwater, Colin Smith, Chris-Anne McKenzie, Simon R. Cox, Ian J. Deary and Tara L. Spires-Jones
    Citation: Acta Neuropathologica Communications 2015 3:53

    The Erratum to this article has been published in Acta Neuropathologica Communications 2015 3:83

  40. Numerous pathogenic mutations responsible for mitochondrial diseases have been identified in mitochondrial DNA (mtDNA)-encoded tRNA genes. In most cases, however, the detailed molecular pathomechanisms and cel...

    Authors: Hideyuki Hatakeyama, Ayako Katayama, Hirofumi Komaki, Ichizo Nishino and Yu-ichi Goto
    Citation: Acta Neuropathologica Communications 2015 3:52
  41. Synaptic dysfunction and intracellular transport defects are early events in Alzheimer’s disease (AD). Extracellular amyloid β (Aβ) oligomers cause spine alterations and impede the transport of proteins and or...

    Authors: Tomohiro Umeda, Elisa M. Ramser, Minato Yamashita, Koichi Nakajima, Hiroshi Mori, Michael A. Silverman and Takami Tomiyama
    Citation: Acta Neuropathologica Communications 2015 3:51

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:7

  42. The term atypical pituitary adenoma (APA) was revised in the 2004 World Health Organization (WHO) classification of pituitary tumors. However, two of the four parameters required for the diagnosis of APAs were fo...

    Authors: Christian P. Miermeister, Stephan Petersenn, Michael Buchfelder, Rudolf Fahlbusch, Dieter K. Lüdecke, Annett Hölsken, Markus Bergmann, Hans Ulrich Knappe, Volkmar H. Hans, Jörg Flitsch, Wolfgang Saeger and Rolf Buslei
    Citation: Acta Neuropathologica Communications 2015 3:50

    The Erratum to this article has been published in Acta Neuropathologica Communications 2016 4:21

  43. Cerebral microbleeds correspond to blood breakdown products, including hemosiderin-containing macrophages around small vessels on histological examination. Superficial lobar cerebral microbleeds are increasing...

    Authors: Enikö Kövari, Andreas Charidimou, François R. Herrmann, Panteleimon Giannakopoulos, Constantin Bouras and Gabriel Gold
    Citation: Acta Neuropathologica Communications 2015 3:49
  44. Tuberous sclerosis complex (TSC) is a genetic disease resulting from mutation in TSC1 or TSC2 and subsequent hyperactivation of mammalian Target of Rapamycin (mTOR). Common TSC features include brain lesions, suc...

    Authors: Anna R. Malik, Ewa Liszewska, Agnieszka Skalecka, Malgorzata Urbanska, Anand M. Iyer, Lukasz J. Swiech, Malgorzata Perycz, Kamil Parobczak, Patrycja Pietruszka, Malgorzata M. Zarebska, Matylda Macias, Katarzyna Kotulska, Julita Borkowska, Wieslawa Grajkowska, Magdalena E. Tyburczy, Sergiusz Jozwiak…
    Citation: Acta Neuropathologica Communications 2015 3:48
  45. The progression of amyotrophic lateral sclerosis (ALS) through the brain has recently been staged using independent neuropathological and neuroimaging modalities. The two schemes tie into the concept of pathol...

    Authors: Manaal Fatima, Rachel Tan, Glenda M. Halliday and Jillian J. Kril
    Citation: Acta Neuropathologica Communications 2015 3:47
  46. The inverse correlation between prevalence of auto-immune disorders like the chronic neuro-inflammatory disease multiple sclerosis (MS) and the occurrence of helminth (worm) infections, suggests that the helmi...

    Authors: Gijs Kooij, Rens Braster, Jasper J. Koning, Lisa C. Laan, Sandra J. van Vliet, Tamara Los, Anne Marieke Eveleens, Susanne M. A. van der Pol, Elisabeth Förster-Waldl, Kaan Boztug, Alexandre Belot, Katka Szilagyi, Timo K. van den Berg, Jaap D. van Buul, Marjolein van Egmond, Helga E. de Vries…
    Citation: Acta Neuropathologica Communications 2015 3:45
  47. Protein aggregation is a common cause of neuropathology. The protein aggregation myopathy Limb-Girdle Muscular Dystrophy 1D (LGMD1D) is caused by mutations of amino acids Phe89 or Phe93 of DNAJB6, a co-chapero...

    Authors: Alessandra Ruggieri, Francesco Brancati, Simona Zanotti, Lorenzo Maggi, Maria Barbara Pasanisi, Simona Saredi, Chiara Terracciano, Carlo Antozzi, Maria Rosaria D′Apice, Federica Sangiuolo, Giuseppe Novelli, Christian R. Marshall, Stephen W. Scherer, Lucia Morandi, Luca Federici, Roberto Massa…
    Citation: Acta Neuropathologica Communications 2015 3:44

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