|
Category
|
Numbers
|
Age @ Onset
|
Disease Duration
|
Age @ Surgery
|
|---|
|
HS
|
2144 (31.2%)
|
11,4
|
22,7
|
34,1
|
|
Dual
|
262 (3.9%)
|
8,6
|
14,6
|
23,3
|
|
Tumors
|
1680 (25.9%)
|
15,4
|
11,5
|
26,8
|
|
Malformations
|
1238 (18.3%)
|
6,0
|
12,1
|
18,3
|
|
No Lesion
|
542 (8%)
|
11,9
|
15,0
|
27,9
|
|
Vascular
|
369 (5.5%)
|
23,1
|
12,7
|
35,9
|
|
Scars
|
344 (5.1%)
|
9,7
|
14,9
|
25,3
|
|
Encephalitis
|
138 (2%)
|
13,2
|
7,7
|
20,7
|
|
Double
|
30 (0.4%)
|
7,0
|
14,8
|
21,4
|
|
Total
|
6747
|
11,8
|
16,1
|
27,9
|
- HS-Hippocampal sclerosis; Dual-dual pathology includes HS with any other principle lesion such as tumors, malformations of cortical development (excluding associated FCD Type IIIA according to the ILAE classification of 2013); vascular malformations, glial scars (excluding postsurgical scars), or encephalitis; Tumors see Table 2; Malformations of cortical development include Focal Cortical Dysplasia (ILAE classification of 2011), polymicrogyria, hemimegalencephaly, hypothalamic hamartoma or cortical tubers; No lesion – microscopic inspection of surgical sample could not reveal any specific lesion entity, including no-HS and gliosis only; Vascular malformations include cavernoma and meningoangiomatosis in Sturge-Weber syndrome, but not ischemic stroke or hypertensive hemorrhages; glial or glio-mesodermal scars include traumatic brain injury and any pre−/peri- or postnatal stroke lesion, excluding postsurgical scaring; Encephalitis includes Rasmussen, limbic or other focal infection; Double pathology represents a combination of at least 2 principal lesions, excluding HS; Age at onset, duration of epilepsy and age at surgery in years
