Fig. 6From: Increased soluble amyloid-beta causes early aberrant brain network hypersynchronisation in a mature-onset mouse model of amyloidosisRepresentative images of ex vivo amyloid pathology. Overview showing the absence of amyloid β plaque load (Thioflavin-S, green) in the TG mice at 8w post DOX treatment and their presence in the cortical and hippocampal regions 28w post DOX treatment, as compared to age-matched control mice. Scale bar, 275 μmBack to article page