Skip to main content
Fig. 6 | Acta Neuropathologica Communications

Fig. 6

From: Complement 3+-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia

Fig. 6

GLP-1R is upregulated in TKO-mice but not in WT-mice after prion infection a Western blot analysis of GLP-1R at clinical prion disease and age-matched controls (n = 3 individual mice per group). b Quantification of GLP-1R levels normalized to β-actin showed that GLP-1R is significantly increased in TKO-mice at clinical prion disease (p = 0.0001); n = 3 independent animals per group. c Western blot analysis of GLP-1R at day 80 p.i. and age-matched controls (n = 3 individual mice per group). d Quantification of GLP-1R levels normalized to β-actin showed that GLP-1R is increased in TKO-mice at day 80 post infection, but significantly downregulated in WT-mice (p = 0.0022); n = 3 independent animals per group

Back to article page