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Table 1 Cohort characteristics

From: Different curcumin forms selectively bind fibrillar amyloid beta in post mortem Alzheimer’s disease brains: Implications for in-vivo diagnostics

#

PM delay (h:min)

Pathological diagnosis (mutation)

Sex

Age

Braak

Amyloid

1

6:35

Control

F

92

III

0

2

7:10

Control

F

78

I

A

3

4:35

Control

F

78

II

A

4

7:15

Control

M

95

II

B

5

5:15

Control

M

83

I

A

6

5:00

EOAD

M

61

VI

C

7

5:05

EOAD

M

59

VI

C

8

4:40

EOAD

M

62

V

B

9

4:45

EOAD

M

64

V

C

10

5:15

EOAD

M

62

VI

C

11

5:30

LOAD

M

88

VI

C

12

7:00

LOAD

F

92

V

C

13

4:40

LOAD

F

89

V

C

14

6:25

LOAD

F

91

IV

C

15

3:05

LOAD

M

74

VI

C

16

6:05

CAA type 1

M

68

II

C

17

4:20

CAA type 1

M

81

V

C

18

4:20

CAA type 1

F

96

V

C

19

3:25

CAA type 1

M

94

V

C

20

6:00

CAA type 1

F

75

V

C

21

5:30

PART

F

81

II

0

22

3:52

PART

F

89

III

0

23

5:00

PART

F

87

II

0

24

5:50

PART

F

93

II

0

25

6:35

PART

F

103

IV

0

26

3:35

FTLD-TDP (Progranulin)

F

76

n.a.

A

27

5:23

FTLD-Tau (P301L)

M

60

n.a.

0

28

6:25

FTLD-Tau (P301L)

M

64

n.a.

0

29

6:15

FTLD-Tau (PiD)

M

60

n.a.

0

30

24:00

PD

M

57

I

0

31

33:00

LBD/AD

F

78

VI

B

32

14:00

PD

M

68

I

0

  1. Abbreviations: CAA type 1 capillary cerebral amyloid angiopathy type 1, EOAD early onset Alzheimer’s disease, FTLD-tau frontotemporal lobar degeneration with tau pathology, FTLD-TDP-43 frontotemporal lobar degeneration with TDP-43 pathology, LOAD late onset Alzheimer’s disease, n.a. not applicable, PART primary age-related tauopathy, PiD Pick’s disease, PM delay post-mortem delay and PD Parkinson’s disease