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Table 1 Cohort characteristics

From: Different curcumin forms selectively bind fibrillar amyloid beta in post mortem Alzheimer’s disease brains: Implications for in-vivo diagnostics

# PM delay (h:min) Pathological diagnosis (mutation) Sex Age Braak Amyloid
1 6:35 Control F 92 III 0
2 7:10 Control F 78 I A
3 4:35 Control F 78 II A
4 7:15 Control M 95 II B
5 5:15 Control M 83 I A
6 5:00 EOAD M 61 VI C
7 5:05 EOAD M 59 VI C
8 4:40 EOAD M 62 V B
9 4:45 EOAD M 64 V C
10 5:15 EOAD M 62 VI C
11 5:30 LOAD M 88 VI C
12 7:00 LOAD F 92 V C
13 4:40 LOAD F 89 V C
14 6:25 LOAD F 91 IV C
15 3:05 LOAD M 74 VI C
16 6:05 CAA type 1 M 68 II C
17 4:20 CAA type 1 M 81 V C
18 4:20 CAA type 1 F 96 V C
19 3:25 CAA type 1 M 94 V C
20 6:00 CAA type 1 F 75 V C
21 5:30 PART F 81 II 0
22 3:52 PART F 89 III 0
23 5:00 PART F 87 II 0
24 5:50 PART F 93 II 0
25 6:35 PART F 103 IV 0
26 3:35 FTLD-TDP (Progranulin) F 76 n.a. A
27 5:23 FTLD-Tau (P301L) M 60 n.a. 0
28 6:25 FTLD-Tau (P301L) M 64 n.a. 0
29 6:15 FTLD-Tau (PiD) M 60 n.a. 0
30 24:00 PD M 57 I 0
31 33:00 LBD/AD F 78 VI B
32 14:00 PD M 68 I 0
  1. Abbreviations: CAA type 1 capillary cerebral amyloid angiopathy type 1, EOAD early onset Alzheimer’s disease, FTLD-tau frontotemporal lobar degeneration with tau pathology, FTLD-TDP-43 frontotemporal lobar degeneration with TDP-43 pathology, LOAD late onset Alzheimer’s disease, n.a. not applicable, PART primary age-related tauopathy, PiD Pick’s disease, PM delay post-mortem delay and PD Parkinson’s disease